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Page Title: TRAUMATIC CONDITIONS OF THE CENTRAL NERVOUS SYSTEM (CNS)
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PSYCHIATRIC  DISORDERS

aspiration of saliva and mucus. After the attack, administer anticonvulsants, such as barbiturates or  Dilantin®.  The  objective  of  drug  therapy  is complete   suppression   of   symptoms.   Refer   the patient  for  evaluation. l Petit Mal Seizure—This type of seizure is characterized   by   myoclonic   jerks   (shocklike contractions of all or a part of a muscle or group of muscles), akinetic seizures (abnormal absence of  muscular  movement  or  loss  of  muscle  tone), and  sudden  clouding  of  consciousness  for  a  few seconds.  Also,  the  patient  will  normally  not  fall down. The classical symptoms of a sudden vacant expression,  cessation  of  motor  activity,  and  loss of  muscle  tone  are  almost  always  present.  This condition is very common in children but almost never  occurs  after  the  age  of  20.  Activity  will resume abruptly. The patient may experience as many  as  100  attacks  per  day. TREATMENT—Administer  phenobarbital  or other   anticonvulsants.   Refer   the   patient   for evaluation. Cerebrovascular Accident Strokes  are  caused  by  destruction  of  brain matter by intracerebral hemorrhage, thrombosis, embolism,  or  vacular  insufficiency. SYMPTOMS—They  include  headache,  nau- sea, vomiting, convulsions, and coma. Conscious- ness  may  not  always  be  altered.  The  patient may  experience  speech  disturbances,  confusion, loss  of  memory,  reduction  of  sensation,  and paralysis  of  extremities  or  of  a  complete  side  of the body. The onset may be sudden and violent, with the patient falling into an immediate coma and exhibiting stertorous breathing, Death from serious strokes may result in a few minutes to a few  days. TREATMENT—Administer  IV  fluids,  and place the patient on immediate and strict bed rest. Evacuate   the   patient   for   hospitalization immediately. Subarachnoid Hemorrhage This is characterized by sudden bleeding into the subarachnoid space that maybe the result of trauma  or  a  ruptured  aneurysm. SYMPTOMS—Before the aneurysm ruptures, it  may  apply  pressure  to  nerves  that  will  manifest as headaches, ocular palsies, diplopia, squint and facial  pain,  and  a  diminished  visual  field.  After rupture,  severe  headache,  nausea,  vomiting, stiffness of the neck, positive Kernig’s sign, and bilateral  Babinski’s  reflex  are  usually  present.  The consciousness  of  the  patient  may  or  may  not  be affected, and the blood pressure is often elevated. TREATMENT—Keep the patient at rest and maintain  a  fluid  balance,  avoid  opiates  and anticoagulants, and  evacuate  the  patient immediately. TRAUMATIC   CONDITIONS OF  THE  CENTRAL  NERVOUS SYSTEM (CNS) Head Injuries Head  injury  is  the  most  common  of  the traumatic  conditions  of  the  CNS.  These  may  be open or closed, and in each case of head injury, a  neurological  evaluation  should  be  performed. l  Concussion—This  is  the  most  common form of head injury and maybe diagnosed by an altered  state  of  consciousness;  abnormal  vital signs;   bleeding   from   the   ears   and   nose;   con- vulsions;  and  altered  pupillary  reactions.  The patient  normally  recovers  with  no  permanent darnage;  however,  recurrent  concussion  may  cause permanent  damage. Extramural  hematoma  is  hemorrhage  into  the extramural  spaces.    This  condition  is  a  rare occurrence.  The  patient  will  suffer  a  loss  of consciousness  at  the  time  of  the  injury  and eventual  coma  with  several  hours  of  lucidity  in between. While lucid, the patient will exhibit signs of   increased   intracranial   pressure,   such   as headache,   irritability   and   mental   confusion, variations  in  level  of  consciousness,  and hemiplegia  (paralysis  on  one  side  of  the  body). The condition will deteriorate and death will result if  the  problem  if  not  corrected. Subdural hematoma is caused by the rupture of a cerebral vein. There will normally be a loss of consciousness  at  the  time  of  the  injury  followed by  an  asyrnptomatic  period  that  may  last  for several days or weeks. Later the patient may have symptoms  of  increased  intracranial  pressure  as described  above.  About  one  half  of  all  persons 2-30

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